Histone methyltransferases Setd1b increases H3K4me3 level to
Por um escritor misterioso
Descrição
The role of histone modifications: from neurodevelopment to neurodiseases
Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes
Methylation of histone H3 lysine 4 is required for maintenance of beta cell function in adult mice
The Unexpected Noncatalytic Roles of Histone Modifiers in Development and Disease: Trends in Genetics
Histone methyltransferase and histone methylation in inflammatory T-cell responses
Frontiers Histone Methylation of H3K4 Involved in the Anorexia of Carnivorous Mandarin Fish (Siniperca chuatsi) After Feeding on a Carbohydrate-Rich Diet
Frontiers Histone modification and histone modification-targeted anti-cancer drugs in breast cancer: Fundamentals and beyond
Epigenetic regulation of development by histone lysine methylation
The H3K4 methyltransferase Setd1b is essential for hematopoietic stem and progenitor cell homeostasis in mice
Proposed mechanism for ROS-mediated inactivation of H3K4
Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes
High‑level SETD1B gene expression is associated with unfavorable prognosis in hepatocellular carcinoma
de
por adulto (o preço varia de acordo com o tamanho do grupo)
