Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
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Frontiers Metabolic Reprogramming and Reactive Oxygen Species in T Cell Immunity
Mitochondrial reactive oxygen species enable proinflammatory signaling through disulfide linkage of NEMO
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Journal of Cellular Physiology, Cell Biology Journal
Frontiers Mitochondria, Oxidative Stress and Innate Immunity
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
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